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Gingival Diseases

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1. Localized gingivitis is confined to: an area or few teeth.
   
2. Generalized gingivitis affects: the entire mouth.
   
3. Marginal gingivitis affects: marginal gingiva.
   
4. Papillary gingivitis involves: Papilla.
   
5. Papillary gingivitis is one of the earliest signs of: gingivitis.
   
6. Diffuse gingivitis is: throughout the gingiva.
   
7. What are is more frequently involved than any other area? Papilla.
   
8. Example of localized marginal gingivitis: #9 marginal gingivitis.
   
9. Example of generalized marginal, papillary gingivitis: the marginal gingiva and papilla in entire mouth is affected.
   
10. An amalgam tattoo is systemic, you just see it.
    
11. What are 2 of the earliest symptoms of gingival inflammation: BOP & increased gingival fluid (exudates).
    
12. BOP is more objective because: capillaries enlarge, epithelium thins with in pocket causing micro ulcerations.
    
13. Healthy gingiva s what color? Coral pink.
    
14.  More blood flow creates redness and loss of: keratinization.
    
15. Early gingivitis the gingiva is what color? Bright red. Erythema.
    
16. Late gingivitis the gingiva is what color? Paler.
    
17. Why is the gingiva paler and fibrotic in late gingivitis? Because the body is trying to heal itself.
    
18. Metallic pigmentations in the gingiva are caused by: absorption of heavy metals, usually seen in people who have been treated with bismuth or arsenic.
    
19. Where do metallic pigmentations accumulate? In areas of inflammation.
    
20. Melanin is more pigmentation in the gingiva, who is it seen most in? People with darker skin.
    
21. T/F If you have stippling and lose it, it is usually a sign of disease.
    
22. Stillmans clefts: apostrophe indents on marginal gingiva, usually on facial.
    
23. McCalls festunes: cuspids, premolars. Lifesaver shaped on marginal gingiva.
    

24. Gingival recession:  apical migration of gingiva.
    

25.  Recession tells: where the gingiva is.
    

26. Apparent recession: CEJ to marginal gingiva. Just what u see.
    

27. Actual recession: CEJ to base of pocket. Most important.
    

28.  List some of the causes of gingival recession: aging, faulty tooth brushing, tooth position, high frenum attachment, inflammation, trauma.
    

29.  People with recession have a higher risk for: root sensitivity & root caries.
    

30. The main objective in treatment of recession is to: establish attached gingiva, cover up area of recession.
    

31. Lateral positioned flap is used to: move good attached gingiva to cover recession of a few teeth.
    

32. Free gingival autograft: Self.
    

33. Free Gingival allograft: same species.
    

34. Free Gingival zenograft: get from another species (pig).
    

35. Inflammation of gingiva creates: pseudo pockets.
    

36. Gingival Abscess-acute inflammatory response: usually due to foreign body. Eg: toothbrush bristle embedded in gingiva.
    

37. Dilatin Hyperplasia (phenytonin) is: fibrotic & long-term. Can be plaque free but plaque will make it worse.
    

38. ANUG is AKA: Acute necrotizing gingivitis, trench mouth, Vincent’s infection.
    

39. Who is more likely to get ANUG? Stressed, smoking, female, psycho, with poor OH.
    

40. List the clinical features of ANUG: Punched out papilla, pseudomembrane, odor, pain, fetid odor.
    

41.  What bacteria are prevalent in ANUG? Vincenti Borrelia, Treponema Denticola.
    

42. Is ANUG communicable? NO.
    

43. Acute herpetic Gingivostomatitis: 1^st time infected 1%, 99% sub clinical, herpes simplex, usually children, communicable, vesicleàruptureà ulcer.
    

44. Leukemic Gingivitis: immune response lowered. Pale, papilla enlarged.

 Disclaimer: These notes were copied and pasted from files sent to me by Andrea.  They have not been reviewed for errors. You are responsible for checking out the information to verify the accuracy. This site, Amy Nieves and Andrea are not responsible for typographical errors.